Hypothyroidism, estrogen excess, free unsaturated fats cause increased vascular permeability and brain edema, protein leakage, and alteration of the matrix
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Argument #94643c27 1 0 2
If it is true that...
Imbalances of other steroids, including cortisol and DHEA, develop as cells compensate for pregnenolone deficiency 1 0 2and
Hypothyroidism, estrogen excess, free unsaturated fats cause increased vascular permeability and brain edema, protein leakage, and alteration of the matrix 1 0 2Then it must be true that...
Alzheimer-type dementia is different from other dementias, but it overlaps with them, and with age-related and stress-related changes in other organs 1 0 2Mentions
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Estrogen is closely associated with the general loss of fat-free tissue with aging 1 0 2Estrogen increases and acts through the inflammatory mediators, serotonin and histamine, to increase vascular leakiness 1 0 2Free fatty acids and lactate impair glucose use, and promote edema, especially in the lungs 1 0 2Thyroid, progesterone, magnesium, glucose, and saturated fatty acids are among the central protective elements 1 0 2The toxicity of estrogen and unsaturated fats has been recognized for most of the twentieth century 1 0 2Interactions between estrogen and unsaturated fats contribute to the aging process 1 0 2Estrogen and unsaturated fats can interact in ways that change restraint and adaptation into sudden self-destruction 1 0 2Dietary polyunsaturated fatty acids (PUFAs) increase estrogen and decrease other signs of estrogen dominance 1 0 2When both thyroid and progesterone are deficient, even fat cells synthesize estrogen 1 0 2Microtubule transport for Apo E, transthyretin, thyroid, and cholesterol for pregnenolone synthesis is disrupted when estrogen increases 1 0 2