Interactions between estrogen and unsaturated fats contribute to the aging process
Negation: Interactions between estrogen and unsaturated fats do not contribute to the aging process 0 0 0
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7 Consequences
2 Mentions
Arguments
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Opposing Arguments
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Consequences
Argument #0c061b71 1 0 2
If it is true that...
Interactions between estrogen and unsaturated fats contribute to the aging process 1 0 2and
Exposure to estrogen in middle-age increases the risk of Alzheimer's disease in old age 1 0 2Then it must be true that...
Alzheimer-type dementia is different from other dementias, but it overlaps with them, and with age-related and stress-related changes in other organs 1 0 2Argument #3a6c29d6 1 0 2
If it is true that...
The toxicity of estrogen and unsaturated fats has been recognized for most of the twentieth century 1 0 2and
Interactions between estrogen and unsaturated fats contribute to the aging process 1 0 2Then it must be true that...
The effects of unsaturated fats can lead to lower energy production, reduced function, and death 1 0 2Argument #527ea311 1 0 2
If it is true that...
The toxicity of estrogen and unsaturated fats has been recognized for most of the twentieth century 1 0 2and
Interactions between estrogen and unsaturated fats contribute to the aging process 1 0 2and
Excessive supply of unsaturated fats in the diet can override some adaptive, protective regulatory processes 1 0 2and
Unsaturated fats can overcome cholesterol, pregnenolone, and progesterone 1 0 2and
Unsaturated fats, interacting with an excess of iron and a deficiency of oxygen or usable energy, redirect our developmental path 1 0 2Then it must be true that...
Degenerative aging seems to be produced situationally, by various types of contamination of our energy supply 1 0 2Argument #ac84f34d 1 0 2
If it is true that...
Interactions between estrogen and unsaturated fats contribute to the aging process 1 0 2and
Excessive supply of unsaturated fats in the diet can override some adaptive, protective regulatory processes 1 0 2Then it must be true that...
The effects of unsaturated fats can lead to lower energy production, reduced function, and death 1 0 2Argument #bc346bb7 1 0 2
If it is true that...
The toxicity of estrogen and unsaturated fats has been recognized for most of the twentieth century 1 0 2and
Interactions between estrogen and unsaturated fats contribute to the aging process 1 0 2Then it must be true that...
Unsaturated fats, interacting with an excess of iron and a deficiency of oxygen or usable energy, redirect our developmental path 1 0 2Argument #cdc40daa 1 0 2
If it is true that...
The toxicity of estrogen and unsaturated fats has been recognized for most of the twentieth century 1 0 2and
Interactions between estrogen and unsaturated fats contribute to the aging process 1 0 2Then it must be true that...
The human body is a dynamic interaction of cellular trophic influences which govern both form and function 1 0 2Mentions
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Related Propositions
Unpleasant consequences of estrogen excess resemble some events of aging 1 0 2The absolute levels of estrogen, or the ratio of estrogen to the antiestrogens, increases with aging in a wide variety of organisms of both sexes, including humans 1 0 2Estrogen is closely associated with the general loss of fat-free tissue with aging 1 0 2Estrogen affects our energetics and structure, and how those processes relate to aging, atrophy, cancerization, etc 1 0 2The toxicity of estrogen and unsaturated fats has been recognized for most of the twentieth century 1 0 2Excessive supply of unsaturated fats in the diet can override some adaptive, protective regulatory processes 1 0 2Estrogen and unsaturated fats can interact in ways that change restraint and adaptation into sudden self-destruction 1 0 2Exposure to estrogen in middle-age increases the risk of Alzheimer's disease in old age 1 0 2Dietary polyunsaturated fatty acids (PUFAs) increase estrogen and decrease other signs of estrogen dominance 1 0 2Hypothyroidism, estrogen excess, free unsaturated fats cause increased vascular permeability and brain edema, protein leakage, and alteration of the matrix 1 0 2