Estrogen's stimulation of non-mitochondrial oxygen consumption with the production of lactic acid stimulates blood vessel formation

Negation: Estrogen's stimulation of non-mitochondrial oxygen consumption with the production of lactic acid does not stimulate blood vessel formation 0 0 0

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Argument #61edd97c 1 0 2

If it is true that...

Estrogen's stimulation of non-mitochondrial oxygen consumption with the production of lactic acid stimulates blood vessel formation 1 0 2

and

Progesterone, by increasing oxidative efficiency, opposes this "angiogenic" (neovascularization) effect of estrogen 1 0 2

Then it must be true that...

Estrogen excess prevents the increase of stroke volume as the speed increases, but progesterone increases the stroke volume as the heart accelerates 1 0 2

Mentions

Ray Peat/Aging, estrogen, and progesterone

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Related Propositions

Unpleasant consequences of estrogen excess resemble some events of aging 1 0 2 Estrogen steals oxygen from mitochondria, shifting patterns of growth and adaptation 1 0 2 Stimulation by estrogen doesn't produce the normal amount of carbon dioxide, leading to tissue experiencing oxygen deprivation, swelling, and cell division 1 0 2 All maladies caused by estrogen excess develop in the same way that it interferes with pregnancy, by driving the tissue to require more energy and oxygen than can be delivered to it 1 0 2 Estrogen creates an oxygen deficiency, stimulates first swelling, and then collagen synthesis 1 0 2 Estrogen increases the blood flow to particular organs, but apparently less than it increases their oxygen demand 1 0 2 Progesterone, by increasing oxidative efficiency, opposes this "angiogenic" (neovascularization) effect of estrogen 1 0 2 The electron transfer process of the mitochondria is interrupted by the futile redox cycling catalyzed by estrogens 1 0 2 Estrogen increases and acts through the inflammatory mediators, serotonin and histamine, to increase vascular leakiness 1 0 2 Estrogen affects our energetics and structure, and how those processes relate to aging, atrophy, cancerization, etc 1 0 2