Increased calcium uptake is not associated with lipid peroxidation and cell death
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Stress, a "respiratory defect", and free radical damage are common factors in disease and aging 1 0 2Melatonin can act as a pro-oxidant at physiologically relevant concentrations 1 0 2In the aged brain, glial cells multiply while neurons die 1 0 2A deficiency of polyunsaturated fatty acids leads to altered rates of cellular regeneration and differentiation 1 0 2The increased lipid peroxidation of old age represents a vicious circle, in which the loss of the antioxidants and vitamin A leads to their further destruction 1 0 1The mitochondrial energy problem, cytochrome oxidase and its regulation; body temperature/pulse-rate cycle disturbance; lipid peroxidation; respiratory defect; altered amino acid uptake; memory impairment; dominance of the excitatory systems vs. the inhibitory adenosine/GABA/progesterone/pregnenolone system are functional and biochemical observations of Alzheimer's disease 1 0 1Increased calcium uptake is associated with lipid peroxidation and cell death 1 0 1Estrogen and calcium depolymerize microtubules 1 0 2Free radicals and calcium cause multiple cell injuries including nerve-death 1 0 2The random production of free radicals acts as a signalling process on a strictly physiological level 1 0 2