Endotoxin does not impair mitochondria, does not increase estrogen levels, and does not cause Kupffer cells in the liver to produce more tumor necrosis factor
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Estrogen steals oxygen from mitochondria, shifting patterns of growth and adaptation 1 0 2Stimulation by estrogen doesn't produce the normal amount of carbon dioxide, leading to tissue experiencing oxygen deprivation, swelling, and cell division 1 0 2Estrogen's stimulation of non-mitochondrial oxygen consumption with the production of lactic acid stimulates blood vessel formation 1 0 2The electron transfer process of the mitochondria is interrupted by the futile redox cycling catalyzed by estrogens 1 0 2Cells of the immune system release tumor necrosis factor and other inflammatory cytokines, and the bowel becomes more permeable, allowing endotoxin and even bacteria to enter the blood 1 0 2Endotoxin impairs mitochondria, increases estrogen levels, causes Kupffer cells in the liver to produce more tumor necrosis factor 1 0 2Despite its name, tumor necrosis factor stimulates the growth and metastasis of some types of cancer 1 0 2Increased estrogen, nitric oxide, excitatory amino acids, cortisol, lactate, free unsaturated fatty acids, prolactin, growth hormone, histamine, serotonin, tumor necrosis factor and other pro-inflammatory cytokines and kinins, and a variety of prostaglandins and eicosanoids have been identified as anti-mitochondrial, anti-respiratory agents 1 0 2Endotoxemia may be a factor in nerve degeneration only during adult life, but it is sometimes present during pregnancy 1 0 2Endotoxin poisoning eventually tends to intervene during stress and aging, exacerbating the trend begun under the influence of other factors 1 0 2